Paper

Effect of high glucose on cytokine production by human peripheral blood immune cells and type I interferon signaling in monocytes: Implications for the role of hyperglycemia in the diabetes inflammatory process and host defense against infection.

Published Oct 1, 2018 · Ronghua Hu, C. Xia, E. Butfiloski

Clinical immunology

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Abstract

Abstract removed due to Elsevier request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.

Study Snapshot

Key takeawayHigh glucose levels can promote inflammation in diabetes by promoting proinflammatory cytokines and impair host defense against microbial infections by suppressing type I interferon production and signaling.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

Effect of high glucose on cytokine production by human peripheral blood immune cells and type I interferon signaling in monocytes: Implications for the role of hyperglycemia in the diabetes inflammatory process and host defense against infection.

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