Paper
Hemodynamic effects of 3-hydroxytyramine (dopamine) in experimentally induced shock.
Published Feb 1, 1969 · Marciano Carvalho, J. Vyden, Harold Bernstein
The American journal of cardiology
Q1 SJR score
22
Citations
0
Influential Citations
Abstract
Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.
Study Snapshot
3-hydroxytyramine effectively restores systemic pressure and increases blood flow in cardiogenic, traumatic, and hemorrhagic shock states, likely due to its vasodilating and pressor effects on both alpha and beta adrenergic receptors.
PopulationOlder adults (50-71 years)
Sample size24
MethodsObservational
OutcomesBody Mass Index projections
ResultsSocial networks mitigate obesity in older groups.
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References
Dopamine in the treatment of hypotension and shock.
Dopamine effectively treats hypotension and shock by increasing myocardial contractility without vasoconstriction, offering a potential alternative to beta-adrenergic amines.
1966·142citations·K. Maccannell et al.·The New England journal of medicine
The New England journal of medicine
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Isoproterenol effectively treats shock due to gram-negative bacteremia, allowing further blood volume expansion with reduced risk of acute circulatory overload.
1966·35citations·G. Kardos·The New England journal of medicine
The New England journal of medicine
Direct Renal Vasodilatation Produced by Dopamine in the Dog
Intravenous dopamine infusion increases renal blood flow and decreases renal vascular resistance in dogs, with unique effects distinct from the conventional beta-adrenergic mechanism.
1965·246citations·J. McNay et al.·Circulation Research
Circulation Research
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Dopamine's pharmacological actions in man are distinct from those of epinephrine and norepinephrine, making it potentially useful in treating patients with inadequate cardiac output.
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Circulation Research
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Dopamine has significant positive inotropic and chronotropic effects in the dog heart-lung preparation, with longer-lasting effects and less cardiac depression.
1962·44citations·J. Holmes et al.·Circulation Research
Circulation Research
Effect of shock and of vasopressor drugs on the regional circulation of the brain, heart, kidney and liver.
Direct continuous measurements of blood flow in vital organs during acute shock and vasopressor therapy provide a more complete understanding of their effects.
1960·103citations·E. Corday et al.·The American journal of medicine
The American journal of medicine
Citations
Fluid Resuscitation Therapy for Hemorrhagic Shock
Hemorrhagic shock is a severe life-threatening emergency, and trauma nurses should understand the various clinical aspects and fluid resuscitation therapy recommendations for this type of shock.
2007·23citations·Joseph R. Spaniol et al.·Journal of Trauma Nursing
Journal of Trauma Nursing
Pressor effects of the α2‐adrenoceptor agonist B‐HT 933 in anaesthetized and haemorrhagic rats: comparison with the haemodynamic effects of amidephrine
BHT 933 can increase blood pressure in anaesthetized rats with haemorrhage, but its effects are less effective than those of amidephrine.
1989·6citations·M. Maclean et al.·British Journal of Pharmacology
British Journal of Pharmacology
Effects of dopamine on canine intrarenal blood flow distribution during hemorrhage.
Dopamine infusion during hemorrhage in dogs improves renal blood flow distribution and may partially explain improved renal function in patients in shock.
1979·21citations·Richard E. Neiberger et al.·Kidney international
Kidney international
Cardiovascular effects of dopamine after experimental coronary occlusion in the dog.
Dopamine can increase heart rate and contractility in dogs with experimental coronary infarction, potentially benefiting shock treatment.
1975·1citation·S. Takeuchi·Japanese heart journal
Japanese heart journal
Effect of Dopamine and Calcium on Lipolysis and Myocardial Ischemic Injury following Acute Coronary Occlusion in the Dog
Dopamine-induced lipolysis significantly contributes to the enlargement of ischemic injury in the myocardium following acute coronary artery occlusion, likely due to metabolic stimulation of myocardial oxygen requirements.
1974·24citations·J. Lekven et al.·Circulation Research
Circulation Research