Paper
Hexachlorobenzene toxicity in the monkey primordial germ cell without induced porphyria.
Published 1993 · John Jarrell, Avril McMahon, D. Villeneuve
Reproductive toxicology
Q2 SJR score
42
Citations
1
Influential Citations
Abstract
Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.
Study Snapshot
Hexachlorobenzene can cause dose-related toxic effects in monkey primordial germ cells without causing systemic or hepatic effects, suggesting a distinct mechanism for ovotoxicity.
PopulationOlder adults (50-71 years)
Sample size24
MethodsObservational
OutcomesBody Mass Index projections
ResultsSocial networks mitigate obesity in older groups.
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References
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Citations
Early-life Farm Exposure and Ovarian Reserve in a US Cohort of Women
Prenatal farm exposure may result in reduced adult ovarian reserve, while childhood farm exposure shows weak or null associations with ovarian reserve.
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Hexachlorobenzene as a persistent organic pollutant: Toxicity and molecular mechanism of action
Chronic exposure to hexachlorobenzene (HCB) can cause a variety of health effects in humans and animals, with molecular mechanisms playing a key role in its toxicity.
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Association of Hexachlorobenzene (HCB), Dichlorodiphenyltrichloroethane (DDT), and Dichlorodiphenyldichloroethylene (DDE) with in Vitro Fertilization (IVF) Outcomes
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