Carbamazepine inhibition of N-methyl-D-aspartate-evoked calcium influx in rat cerebellar granule cells.

doi:10.1016/s0022-3565(25)12243-x

Paper

Carbamazepine inhibition of N-methyl-D-aspartate-evoked calcium influx in rat cerebellar granule cells.

Published Jan 1, 1996 · C. Hough, Robert P. Irwin, Robert P. Irwin

The Journal of pharmacology and experimental therapeutics

Q2 SJR score

78

Citations

0

Influential Citations

Full textSemantic Scholar

Abstract

Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.

In Vitro Study

Highly Cited

Study Snapshot

Carbamazepine effectively blocks NMDA receptor responses in cerebellar granule cells, potentially leading to cerebellar dysfunction and seizures.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

Sign up to use Study Snapshot

Consensus is limited without an account. Create an account or sign in to get more searches and use the Study Snapshot.

Create an account

Paper

Carbamazepine inhibition of N-methyl-D-aspartate-evoked calcium influx in rat cerebellar granule cells.

Sign up to use Study Snapshot

References

Citations

Interaction of Varenicline with Classic Antiseizure Medications in the Mouse Maximal Electroshock-Induced Seizure Model

Varenicline may decrease the protective activity of classic antiseizure medications, potentially causing serious concerns for epileptic tobacco smokers.

The Role of Mitochondria in Mood Disorders: From Physiology to Pathophysiology and to Treatment

Mitochondrial dysfunction is linked to mood disorders, and targeting these pathways with novel therapies could potentially improve treatment outcomes for treatment-resistant patients.

Dysregulation of autism-associated synaptic proteins by psychoactive pharmaceuticals at environmental concentrations

Psychoactive pharmaceuticals at extremely low concentrations can alter the expression of key synaptic proteins, potentially contributing to neurological disorders like ASD by disrupting neuronal development.

A Systematic Review of NMDA Receptor Antagonists for Treatment of Neuropathic Pain in Clinical Practice

NMDAR antagonist agents show potential in treating neuropathic pain, with ketamine showing the most benefit and further investigation needed for further evaluation.

Chronic Treatment with Anti-bipolar Drugs Down-Regulates Gene Expression of TRPC1 in Neurones

Chronic treatment with anti-bipolar drugs like carbamazepine, lithium, and valproic acid leads to down-regulation of TRPC1 gene expression and function in neurones, potentially explaining their action.