Paper
Effects of bromodichloromethane on ex vivo and in vitro luteal function and bromodichloromethane tissue dosimetry in the pregnant F344 rat.
Published Aug 1, 2007 · Susan R. Bielmeier, A. Murr, D. Best
Toxicology in vitro : an international journal published in association with BIBRA
Q2 SJR score
14
Citations
1
Influential Citations
Abstract
Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.
Non-RCT
Animal StudyStudy Snapshot
Bromodichloromethane disrupts pregnancy in F344 rats by disrupting LH secretion and the corpora lutea's ability to respond to LH.
PopulationOlder adults (50-71 years)
Sample size24
MethodsObservational
OutcomesBody Mass Index projections
ResultsSocial networks mitigate obesity in older groups.
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References
Bromodichloromethane inhibits human placental trophoblast differentiation.
Bromodichloromethane disrupts syncytiotrophoblast formation and inhibits chorionic gonadotropin secretion in vitro, potentially impacting adverse pregnancy outcomes.
2004·41citations·Jiangang Chen et al.·Toxicological sciences : an official journal of the Society of Toxicology
Toxicological sciences : an official journal of the Society of Toxicology
Serum hormone characterization and exogeneous hormone rescue of bromodichloromethane-induced pregnancy loss in the F344 rat.
Bromodichloromethane-induced pregnancy loss in rats occurs via an LH-mediated mode of action, and both progesterone and hCG can prevent this loss when administered together.
2003·36citations·Susan R. Bielmeier et al.·Toxicological sciences : an official journal of the Society of Toxicology
Toxicological sciences : an official journal of the Society of Toxicology
Effect of bromodichloromethane on chorionic gonadotrophin secretion by human placental trophoblast cultures.
Bromodichloromethane (BDCM) disrupts chorionic gonadotropin secretion in human placental trophoblast cultures, suggesting it may target the placenta and contribute to adverse pregnancy outcomes.
2003·27citations·Jiangang Chen et al.·Toxicological sciences : an official journal of the Society of Toxicology
Toxicological sciences : an official journal of the Society of Toxicology
Chlorination by-products in drinking water and menstrual cycle function.
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Citations
Impacts of disinfection byproduct exposures on male reproductive health: Current evidence, possible mechanisms and future needs.
Haloacetic acids are male reproductive toxicants, but epidemiological evidence is insufficient to link disinfection byproduct exposures to adverse reproductive outcomes.
2023·7citations·Min Zhang et al.·Chemosphere
Chemosphere
Exposure to disinfection by-products and reproductive hormones among women: Results from the Tongji Reproductive and Environmental (TREE) study.
Urinary DCAA exposure is associated with increased serum progesterone and prolactin levels in women undergoing assisted reproductive technology, while trichloroacetic acid exposure is not.
2022·17citations·Chong Liu et al.·Environmental research
Environmental research
Windows of sensitivity to toxic chemicals in the development of the endocrine system: an analysis of ATSDR’s toxicological profile database
Toxic chemicals can affect endocrine system development at various points, including gestation and lactation, highlighting the need for more developmental toxicity studies on hormone alterations.
2020·2citations·MC Buser et al.·International Journal of Environmental Health Research
International Journal of Environmental Health Research
Disinfection Byproducts in Drinking Water and Evaluation of Potential Health Risks of Long-Term Exposure in Nigeria
THMs in Nigerian drinking water exceed standard maximum contaminant levels, with higher average intake in adults and comparable ingestion in children, potentially increasing long-term cancer risk.
2017·46citations·N. Benson et al.·Journal of Environmental and Public Health
Journal of Environmental and Public Health
Trihalomethanes in public drinking water and stillbirth and low birth weight rates: an intervention study.
Enhanced coagulation (EC) in water treatment did not significantly affect birth outcome rates, but a large decrease in chloroform concentrations was associated with significant reductions in very low birth weight rates.
2014·17citations·N. Iszatt et al.·Environment international
Environment international