The anti-inflammatory effects of ethyl acetate on Lipopolysaccharide/D-galactosamine challenged mice and Lipopolysaccharide activated RAW264.7 cells.

doi:10.1016/j.bbrc.2012.09.088

Paper

DOI: 10.1016/j.bbrc.2012.09.088

The anti-inflammatory effects of ethyl acetate on Lipopolysaccharide/D-galactosamine challenged mice and Lipopolysaccharide activated RAW264.7 cells.

Published Oct 26, 2012 · Tengfei Liu, Nan Xiao, Yuxin Liu

Biochemical and biophysical research communications

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Abstract

Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.

Non-RCT

Animal Study

Study Snapshot

Ethyl acetate has anti-inflammatory properties, protecting mice from LPS-induced endotoxic shock and decreasing inflammation-related mediators in RAW264.7 cells.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

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Short-Chain Fatty Acids Suppress Lipopolysaccharide-Induced Production of Nitric Oxide and Proinflammatory Cytokines Through Inhibition of NF-κB Pathway in RAW264.7 Cells

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