Evidences for CYP3A4 autoactivation in the desulfuration of dimethoate by the human liver.

doi:10.1016/J.TOX.2007.08.081

Paper

Evidences for CYP3A4 autoactivation in the desulfuration of dimethoate by the human liver.

Published Nov 20, 2007 · F. Buratti, E. Testai

Toxicology

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53

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1

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Abstract

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In Vitro Study

Study Snapshot

Dimethoate's atypical kinetic profile in the human liver may explain its more severe symptoms and higher fatality rate than chlorpyrifos, suggesting CYP3A4 inhibitors may be a complementary treatment for DIM-poisoned patients.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

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Evidences for CYP3A4 autoactivation in the desulfuration of dimethoate by the human liver.

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