Mechanism of sodium channel block by venlafaxine in guinea pig ventricular myocytes.

doi:10.1016/s0022-3565(24)35098-0

Paper

Mechanism of sodium channel block by venlafaxine in guinea pig ventricular myocytes.

Published Oct 1, 1999 · Majed Khalifa, P. Daleau, and Jacques Turgeon

The Journal of pharmacology and experimental therapeutics

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105

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Abstract

Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.

In Vitro Study

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Study Snapshot

Venlafaxine blocks sodium channel activity in guinea pig ventricular myocytes after binding to the resting state of the channel, exhibiting different characteristics than those observed with most tricyclic antidepressants or conventional class I antiarrhythmic drugs.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

Mechanism of sodium channel block by venlafaxine in guinea pig ventricular myocytes.

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