Mechanisms of toxicity and tolerance to diisopropylphosphorofluoridate at the neuromuscular junction of the rat.

doi:10.1016/0041-008X(86)90259-0

Paper

DOI: 10.1016/0041-008X(86)90259-0

Mechanisms of toxicity and tolerance to diisopropylphosphorofluoridate at the neuromuscular junction of the rat.

Published Jul 1, 1986 · Ramesh C. Gupta, G. T. Patterson, W. Dettbarn

Toxicology and applied pharmacology

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Abstract

Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.

Non-RCT

Animal Study

Study Snapshot

Neuromuscular adaptation to diisopropylphosphorofluoridate (DFP) involves recovery of acetylcholinesterase activity through de novo synthesis and reduction in nicotinic acetylcholine receptor density.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

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