NG-monomethyl-L-arginine causes nitric oxide synthesis in isolated arterial rings: trouble in paradise.

doi:10.1016/0006-291X(92)91097-A

Paper

NG-monomethyl-L-arginine causes nitric oxide synthesis in isolated arterial rings: trouble in paradise.

Published Oct 30, 1992 · Stephen L. Archer, Václav Hampl

Biochemical and biophysical research communications

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72

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0

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Abstract

Abstract hidden due to publisher request; this does not indicate any issues with the research. Click the full text link above to read the abstract and view the original source.

In Vitro Study

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Study Snapshot

NG-monomethyl-L-arginine is a partial agonist for nitric oxide synthesis in pulmonary arterial and aortic rings, potentially causing vasodilation.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

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Paper

NG-monomethyl-L-arginine causes nitric oxide synthesis in isolated arterial rings: trouble in paradise.

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References

Citations

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Characterization of Local Vascular Effects of the Nitric Oxide Inhibitor NG‐Monomethyl‐L‐Arginine on Dorsal Hand Veins

NG-monomethyl-L-arginine (L-NMMA) increases vein size in healthy males, with venous reactivity likely genetically predetermined.

Effects of the Nitric Oxide Synthase Inhibitor L-NAME on Recognition and Spatial Memory Deficits Produced by Different NMDA Receptor Antagonists in the Rat

L-NAME can counteract recognition and spatial memory deficits caused by NMDA receptor antagonists like MK-801 and ketamine in rats, but also disrupts their spatial memory performance.

Differential sensitivity of basal and acetylcholine‐induced activity of nitric oxide to blockade by asymmetric dimethylarginine in the rat aorta

Asymmetric dimethylarginine (ADMA) selectively inhibits basal but not acetylcholine-induced nitric oxide activity in the rat aorta.

Role of ventral hippocampal nitric oxide/cGMP pathway in anxiety-related behaviors in rats submitted to the elevated T-maze

Nitric oxide in the ventral hippocampus plays a crucial role in modulating defensive behaviors in rats exposed to the elevated T-maze.