Nerve terminal facilitatory action of 4-aminopyridine: An analysis of the rising phase of the endplate potential

doi:10.1016/0028-3908(78)90048-5

Paper

Nerve terminal facilitatory action of 4-aminopyridine: An analysis of the rising phase of the endplate potential

Published Jul 1, 1978 · R. Jacobs, E. S. Burley

Neuropharmacology

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18

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1

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Abstract

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In Vitro Study

Study Snapshot

4-aminopyridine increases endplate potential amplitude and rate of rise in the frog neuromuscular junction, likely due to an increase in quantal release sites.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

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Paper

Nerve terminal facilitatory action of 4-aminopyridine: An analysis of the rising phase of the endplate potential

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References

Citations

Modulation of Spatial Alternation and Anxiety by Septal Scopolamine Systemic Diazepam in Mice

Septal scopolamine and diazepam combined can decrease anxiety and impair spatial alternation in mice with impaired cholinergic activity.

Baclofen produces dose-related working memory impairments after intraseptal injection.

Baclofen impairs working memory in rats, reducing correct choices and increasing errors without affecting navigation or food consumption abilities.

Intraseptal injection of GABA and benzodiazepine receptor ligands alters highaffinity choline transport in the hippocampus

Intraseptal injection of GABA and benzodiazepine receptor ligands alters hippocampal high-affinity choline transport, with potential functional and therapeutic implications.

Effect of 4-aminopyridine on the postsynaptic action of polymyxin B.

4-aminopyridine effectively antagonizes the postsynaptic action of polymyxin B, supporting the receptor desensitization hypothesis.

Suppression by phenytoin of convulsant-induced afterdischarges at presynaptic nerve terminals

Phenytoin effectively suppresses convulsant-induced afterdischarges at presynaptic nerve terminals, making presynaptic nerve terminals a preferred site for both convulsant and anticonvulsant actions.