Tris acetylacetonate aluminium(III) induces osmotic fragility and acanthocyte formation in suspended erythrocytes.

doi:10.1016/0378-4274(89)90154-9

Paper

DOI: 10.1016/0378-4274(89)90154-9

Tris acetylacetonate aluminium(III) induces osmotic fragility and acanthocyte formation in suspended erythrocytes.

Published 1989 · P. Zatta, M. Perazzolo, B. Corain

Toxicology letters

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Abstract

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In Vitro Study

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Tris acetylacetonate aluminium(III) causes osmotic fragility and acanthocyte formation in suspended erythrocytes, with greater effects due to its greater solubility in the cell membrane.

PopulationOlder adults (50-71 years)

Sample size24

MethodsObservational

OutcomesBody Mass Index projections

ResultsSocial networks mitigate obesity in older groups.

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References

DOI: 10.1080/03067318908026853

N-Octanol/Water Partition Coefficients of the Acetylacetonate and Maltolate Complexes of Al(III), Cr(III) and Fe(III) and of Aluminum Lactate

The metal-coordinated ligand's lipophilic-hydrophilic character significantly influences the lipophilicity of M(acac)3 and the hydrophilicity of M(malt)3.

1989·17citations·A. Tapparo et al.·International Journal of Environmental Analytical Chemistry

International Journal of Environmental Analytical Chemistry

DOI: 10.1016/0378-4274(87)90231-1

A neutral lipophilic compound of aluminum(III) as a cause of myocardial infarct in the rabbit.

Acetylacetonate, a stable and moderately lipophilic aluminum(III) complex, can cause severe pathological effects in rabbits, including myocardial infarct.

1987·16citations·P. Zatta et al.·Toxicology letters

Toxicology letters

DOI: 10.1016/0197-4580(86)90102-8

Aluminum and alzheimer's disease

Aluminum accumulation in grey matter in Alzheimer's disease may contribute to its pathogenesis, but its etiological role remains unclear.

1986·209citations·David G. Munoz·Neurobiology of Aging

Neurobiology of Aging

DOI: 10.1016/0197-4580(86)90105-3

Aluminum toxicity among other views on the etiology of Alzheimer disease

Aluminum toxicity is only one of several co-factors in the etiology of Alzheimer's disease, and further research is needed to identify the cause(s).

1986·14citations·Z. Khachaturian·Neurobiology of Aging

Neurobiology of Aging

DOI: 10.1016/0005-2760(85)90113-4

Aluminium salts accelerate peroxidation of membrane lipids stimulated by iron salts.

Aluminium salts accelerate lipid peroxidation stimulated by iron salts at acidic pH, potentially contributing to the increased formation of fluorescent age pigments in the nervous system of toxic aluminum overload patients.

1985·314citations·J. Gutteridge et al.·Biochimica et biophysica acta

Biochimica et biophysica acta

DOI: 10.1002/ana.410080505

Aluminum chloride induced neurofibrillary changes in the developing rabbit: A chronic animal model

Aluminum chloride injection in developing rabbits leads to neurofibrillary changes, but most animals survive for months without clinical symptoms, and some recover without showing clinical signs.

1980·146citations·H. Wiśniewski et al.·Annals of Neurology

Annals of Neurology

Citations

DOI: 10.2478/intox-2019-0007

Aluminium toxicosis: a review of toxic actions and effects

Aluminium toxicosis can cause oxidative stress, immunological alterations, genotoxicity, pro-inflammatory effects, and various diseases, including Alzheimer's, dementia, and sclerosis.

2019·262citations·I. Igbokwe et al.·Interdisciplinary Toxicology

Interdisciplinary Toxicology

DOI: 10.4314/SOKJVS.V16I4.1

A review of the factors that influence erythrocyte osmotic fragility

Intrinsic and extrinsic factors influence erythrocyte osmotic fragility, aiding in laboratory assessment of conditions related to erythrocytes.

2019·29citations·N. Igbokwe·Sokoto Journal of Veterinary Sciences

Sokoto Journal of Veterinary Sciences

DOI: 10.1002/jcb.24026

Oxidative stress due to aluminum exposure induces eryptosis which is prevented by erythropoietin

Chronic aluminum exposure can cause biochemical and morphological alterations in human erythrocytes, and erythropoietin may protect against these effects.

2012·43citations·D. Vota et al.·Journal of Cellular Biochemistry

Journal of Cellular Biochemistry

DOI: 10.1016/S0010-8545(02)00074-7

Aluminium(III) as a promoter of cellular oxidation

Aluminium promotes cellular oxidation, potentially contributing to human pathologies like renal failure and neurodegenerative diseases.

2002·169citations·P. Zatta et al.·Coordination Chemistry Reviews

Coordination Chemistry Reviews

DOI: 10.1016/S0005-2736(01)00427-8

Human erythroid cells are affected by aluminium. Alteration of membrane band 3 protein.

Aluminium exposure can disrupt human erythropoiesis by affecting mature erythrocytes and cellular metabolism in late erythroid progenitors.

2002·39citations·Daniela C Vittori et al.·Biochimica et biophysica acta

Biochimica et biophysica acta